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have identified a novel mechanism by which cocaine promotes activation of D 1 -expressing nAcc neurons, the enhancement of inositol 1,4,5-trisphosphate receptors (IP 3 R) mediated responses via 1 R activation at the endoplasmic reticulum, resulting in augmented Ca 2+ release and amplified depolarization due to subsequent stimulation of transient receptor potential canonical channels (TRPC) [20]

Behavioral time scale synaptic plasticity underlies CA1 place fields

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